Splenius Capitis Syndrome: Muscle Tension Headache

Headache is one of the most
prevalent of all complaints that might bring a patient to seek medical
attention. Diagnostic evaluation of the patient may be simple or comprehensive,
dictated by the clinical situation and patient presentation.  Statistically, however, the most common
etiology of headache is found to be muscle tension in nature.

While the diagnosis of muscle
tension headache might suffice for most circumstances, unless the clinician
locates the precise physical site of the ‘pain generator,’ the chances of
successful treatment are slim.  On the
other hand, treatment is likely to be successful, if diagnosis is refined from
the generic ‘muscle tension headache,’ to the more specific diagnosis.

The most common cause of muscle tension headache (MTH)
results from inflammatory changes at the site of muscular attachment on the
occipital ridge.  In the adult, this
occurs most often at the attachment of the Splenius Capitis and Semispinalis
Capitis Muscles.  As inflammation develops,
entrapment and irritation of Greater Occipital Nerve results. The typical symptom
complex results from muscle spasm as well as from neuralgia.

 

Occipital neuralgia results in a distinct type of pain that
is most commonly characterized by piercing, throbbing, or lancinating  pain in the upper neck, back of the head, and
behind the eyes  Most commonly the
problem is unilateral and because pain in the eye is typical, the mis-diagnosis
of ‘migraine’ is made. The pain of Splenius Capitis Syndrome (SCS) begins in
the neck and then spreads cephalad with referred pain behind the eye.    Anatomic variation is the rule, rather
than the exception, and clinical presentations will therefore vary. 

 

The pain is caused by inflammation, irritation or injury to
the nerves that inntervate the nerve, called the nervi nervorum.  Muscular spasm results in a second pain
constellation. Interestingly, the pain of muscle spasm and the pain that
results from the neuralgia need not occur simultaneously and the nature of the
pain will, therefore, change from episode to episode, adding to clinical
confusion.

 

Causes of Splenius Capitis Syndrome
result of trauma to the back of the head, pinching of the nerves by overly
tight neck muscles, and compression of the nerve as it leaves the spine due to
osteoarthritis.    Localized
inflammation or infection can result in SCS, although this is quite rare.  It is possible that it can be caused by diabetes
(mononeuritis multiplex), and blood vessel inflammation (vasculitis).  Clearly, the most frequent cause is postural,
that is, from prolonged periods of keeping the head in a downward, rotated and
forward position. The muscle tension results in micro trauma to the muscular
attachment, swelling ensues, and myalgia/neuralgia result.  In many cases,
however, no precipitant cause can be found. 

Symptom
Complex

w
Occipital & Shoulder Pain

w
Pain behind the eyes


Awaken with pain

w
Pain looking up.

w
Pain looking down, particularly with rotation.

w
“Migraine” diagnosis is common.

w
Worse with menses, ovulation & salt load.

w
Anticipate sleep disorder.

Causes
of Splenius Capitis Syndrome

w
Trauma of a rotational nature

w
Blunt Trauma, pugilism

w
Flexion/Extension injury

w
Vigorous Cervical Manipulation

w
Surgery


Dental chair


Mastoid Surgery

The causes of SCS can be quite
distant from the site of pain.  That is,
postural problems can result from foot pathology, ankle, knee and hip problems
that result in muscular dystony.  As
occurs in other species, such as the horse physical problems in the lower
extremity can cause physical changes in the axial skeleton. Postural pathology
can result from an arthritic toe or heel spur, resulting in contralateral hip
pain, and same-sided headache.

Injection is performed in a short
series, that is, once or twice with pain relief lasting from a few weeks to
many months.  Skillful injection
technique results in the most satisfactory results, and concurrent use of
common anti-inflammatories speed recovery, particularly when combined with a
low-dose diuretic. Muscle relaxant or anti-convulsants, dosed at bed time
provide relief of the neuralgia and side effects are minimized by the bed-time
administration.

If exacerbation of pain is
triggered by ovulation or dietary salt-load, routine or periodic administration
of a mild diuretic is worthwhile.

David S Klein, MD

David S. Klein, MD, FACA, FACPM was born in Washington, DC, and was raised in Chevy Chase, Maryland. He completed his undergraduate education at the University of Maryland with degrees in Chemistry and Psychology.

Medical School was completed at the University of Maryland at Baltimore, followed by Internship in General Surgery at the University of North Carolina and Residency in Anesthesiology at the Duke University, Durham, North Carolina. Dr Klein has been practicing medicine since 1983, concentrating in Pain Medicine, Minimally Invasive Medicine and Surgery, and Neuroendocrinology. Earning Board Certification in Anesthesiology, Dr. Klein was elected Fellow in the American College of Anesthesiology, and he was elected Fellow in the American College of Pain Medicine. He is currently an adjunct Associate Professor at the University of Central Florida, School of Medicine.

He has focused his private practice on treating patients with hormone imbalance issues, nutritional deficiency related medical problems as well as pain related issues and impairment. With a highly-complex, CLIA licensed laboratory in-house, he has been able to provide rapid-turn around analysis efficiently and cost-effectively.
Lecturing extensively nationally as well as internationally, Dr. Klein has authored many articles on topics relating to pain, injury and nutritionally modulated illness. His radio show, “Pain Free Living,” received top ratings during the 6 years it was on the air. Currently practicing in Longwood, Florida, Dr. Klein practices entirely in the office setting.

Leave a Reply

Your email address will not be published. Required fields are marked *